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These curious vascular abnormalities, occurring in both the cranial and spinal dura, have different presentations at each site; the latter form is discussed with other diseases of the spinal cord in Chap 46 The cranial type is being detected with increasing frequency as re nements continue to be made in imaging of the cerebral vessels, but its true incidence and pathogenesis are not fully known The de ning features are radiologic a nidus of abnormal arteries and veins with arteriovenous shunting contained entirely within the leaflets of the dura The lesion is fed by dural arterial vessels derived from the internal cranial circulation and often, more proli cally, from the external cranial circulation (external carotid artery and muscular branches of the vertebral artery) The venous drainage is complex and is largely into the dural venous sinuses (Fig 34-27) The rapid transit of injected angiographic dye through these stulas accounts for the early opaci cation of the draining venous structures; in the case of high- ow connections, this may not be seen unless images are taken almost immediately after the injection A number of potential feeding vessels must be injected to demonstrate all the conduits into the lesion On CT scanning and MRI, the stula is sometimes detected as a thickening or enhancement of a region barcode add in for word and excel 2013 Excel Add-In for Barcode - Barcode Resource
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Barcode Add in for Word and Excel - Easily generate barcodes in Microsoft Word and Excel with this add-in. The add-in changes the selected data to a barcode ... of dura, generally close to a large dural venous sinus In other cases, the dilated draining vessels may be seen only with the injection of dye or gadolinium Probably, many are not detected by these techniques The origin of these vascular lesions has not been settled several mechanisms may be involved Most evidence suggests that some of these lesions, unlike conventional cerebral AVMs and aneurysms, are not developmental in origin The best-de ned examples are those that arise adjacent to a venous sinus thrombosis or in association with a vascular atresia, most often in the transverse sigmoid sinus or adjacent to the cavernous sinus However, it is not clear whether the abnormality of the dural sinus is the cause or the result of the dural stula In a number of cases, a dural stula has appeared after a forceful head injury, often in a region remote from the site of impact A small group is associated with the Klippel-Trenaunay or Osler-Weber-Rendu syndromes, diseases in which a frequent conjunction with AVMs is well known Usually, all of these causes can be excluded and the largest group remains idiopathic A major obstacle to our understanding of the dural AVM has been the varied and confusing ways in which this lesion presents clinically Subdural hemorrhage is an infrequent mode of presentation, sometimes creating a large and fatal clot; another syndrome is a cerebral-subarachnoid hemorrhage, although this occurs with not nearly the frequency or severity as bleeding from brain AVMs However, our knowledge of the risk of bleeding from dural stulas and the evolution of the lesions is far less precise than it is for cerebral AVMs It appears that the dural lesions most at risk of bleeding are those located in the anterior cranial fossa and at the tentorial incisura Seizures are distinctly uncommon A special syndrome linked to dural AVMs, although it may occur also with high ow cerebral malformations, is of headache, vomiting, and papilledema namely, pseudotumor cerebri (Chap 30) A cranial bruit is infrequent In small children, the high- ow lesions may shunt so much blood as to cause congestive heart failure, similar to malformations of the vein of Galen Treatment is by surgical extirpation or endovascular embolization, a painstaking procedure because of the multitude of poten-. barcode excel 2003 free download Barcode Add-in for Excel for MAC OSX Free Download
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